flydoom

LIF simulation of all 139,255 FAFB-v783 neurons in the model form of Shiu et al. (tau_mem, tau_syn, threshold, reset, refractory, conduction delay), stepped at 0.5 ms. Not L2: nothing writes a synaptic weight during a run. The one plasticity mechanism in the codebase is short-term depression, and it is (a) disabled by default, with the config stating 'The reported model is the frozen one', and (b) a per-neuron presynaptic release resource rather than a per-synapse weight, so even when enabled it is vesicle depletion, not learning. The project's own neuron registry says the punishment-dopamine pathway 'injects into a brain with no plasticity to receive it'. This is a third, independent flydoom rated on its own source; it also reports its own negative result on motion vision in detail, which is good science and is not held against it.